What You Need to Know
CRISPR/Cas9 introduces targeted double‑strand breaks that are repaired via non‑homologous end joining or homology‑directed repair, enabling knockouts or precise substitutions. The edited genes (e.g., SlPPO, GmGlyma.01G123400, FAD2) alter enzymatic pathways that control phenolic oxidation, nitrogen metabolism, and fatty‑acid desaturation. These modifications shift metabolic fluxes, producing lower phenolic oxidation products, higher seed protein, or altered lipid profiles.
The Science
Primary Reaction
CRISPR/Cas9‑mediated double‑strand break and repair